Literally, I can't find it. So frustrating. I was actually going to use it last night. I know, right?
Maddox is almost 17 pounds now! I seriously can't believe it! They told us he'd probably be little forever and would have a hard time gaining weight. I can thank the good chunky baby making genes for that one. Remember how big Bailee was? No? Well before she hit 9 months she was 20 lbs. and she outgrew her infant seat. She was always 95% or off the charts on everything, not just weight. Anyways, Maddox is growing well and I'm grateful.
He LOVES sweet potatoes, carrots, green beans, peas. Will not touch Bananas (he actually gags any time I try and feed him bananas), Peaches, Pears. Will sometimes eat Applesauce, but only the stuff that we made this fall. Doesn't like rice cereal or oatmeal, even with apple juice mixed in. But seriously, give him a veggie and he downs it. Crazy kid. Maddox is drooling about 30 gallons of water an hour, soaks through his clothes, blankets, mommy's clothes. I'm certain he's teething but still hasn't actually cut a tooth. If I were nursing, I'd totally be loving that. He's trying to sit up, hates tummy time, loves his johnny jumper. When I put him on his tummy he pushes with his legs but hasn't yet figured out to push up on his arms. This causes him to rub his face in the ground and let's face it, that's never fun.
Maddox and Bailee ADORE each other. Seriously. If he's asleep she's going in his room all the time to see if he's awake so they can play. When Bailee comes into Maddox's sight, he LIGHTS up. It's so cute. No one can make Maddox smile and giggle like Bailee can. And when she's having a hard time little brother is right there to cheer her up. It can't last forever, though I hope it does, so we'll enjoy it while it does last.
Bailee is so much fun. She is such a girly girl! Can't figure out where that came from. We all know she didn't get it from me. Though she loves being outside, riding her horse, and even getting dirty. Especially if she can do all of that in her princess dress. Last night she was shocked that I know how to saddle a horse. She made me tell her step by step how to do it, and when I was finished she said "You are right! That's exactly how you do it." Funny.
In case you're worried that her birthday isn't planned yet, have no fear. As all good hostess's know, you must plan at least 6 months in advance. She's already given me the details and would like to start on her invites now. So save the date. Funny girl! Bailee loves dance, but wouldn't go this week because she and McKinley were fighting last week. I hope she gets over that, and doesn't let it stop her from doing what she loves. She's too smart for her own good and keeps me on my toes. Love it.
Luke went on his first scout trip as a leader last night. They were cross country skiing into camp, spending the night, playing in the snow, and coming home this evening. He didn't enjoy those activities the first time around, so I'm anxious to hear the report when he gets home. I hope he stayed warm enough and was able to sleep last night.
Well its snack time and nap time for Maddox, and I should probably wake Bailee up and feed her some breakfast.
Saturday, February 6, 2010
Friday, February 5, 2010
Heart: HLHS
Hypoplastic Left Heart Syndrome
(HLHS) is rare but serious. It is the most common
cause of death from heart disease during the first
week of life. With surgical repair or a heart transplant,
about 70% of children born with HLHS live at least 5
years.
In infants born with HLHS, the left ventricle and the
aorta are small and underdeveloped. The left ventricle
is the lower-left chamber of the heart and is
responsible for pumping oxygen-rich blood to the
body. The aorta is the artery that receives oxygen-rich
blood from the left ventricle and sends it through the
body. When the left ventricle and the aorta are too
small, they can't supply the body with enough blood.
Also in HLHS, the mitral and aortic valves are often
narrow or absent. The mitral valve acts like a door that
opens to let blood into the left ventricle. The aortic
valve lets blood out of the left ventricle. Without these
openings, the left ventricle is shut off from the rest of
the heart and the aorta. That means the left ventricle
cannot pump any oxygen-rich blood into the body.
Instead, the oxygen-rich blood returns to the right side
of the heart through an atrial septal defect (ASD), a
hole in the wall that divides the right and left sides of
the heart.
In newborns, the atrial septal defect lets oxygen-rich
blood reach the body because blood can pass from
the right side of the heart and into the aorta, without
going through the left side of the heart. The right side
of the heart pumps blood into the pulmonary artery,
and then a channel called the ductus arteriosus
connects the pulmonary artery to the aorta. The
ductus arteriosus is an important pathway in the fetal
heart, but it closes in the first few days after birth.
Until the ductus closes, oxygen-rich blood enters the
right side of the heart through the atrial septal defect.
The right side of the heart then pumps blood into the
pulmonary artery, and the ductus arteriosus lets blood
flow into the aorta. The aorta carries this oxygen-rich
blood to the body.
This means that for the first few days after birth, the
baby may seem normal because oxygen-rich blood is
reaching the rest of the body. But when that ductus
closes, oxygen-rich blood no longer has a way of
entering the aorta.
When there isn't enough oxygen-rich blood reaching
the body, the baby's skin may turn blue. This is called
cyanosis. Once this happens, the baby needs surgery
or a heart transplant right away.
Taken from Hope for HLHS
(HLHS) is rare but serious. It is the most common
cause of death from heart disease during the first
week of life. With surgical repair or a heart transplant,
about 70% of children born with HLHS live at least 5
years.
In infants born with HLHS, the left ventricle and the
aorta are small and underdeveloped. The left ventricle
is the lower-left chamber of the heart and is
responsible for pumping oxygen-rich blood to the
body. The aorta is the artery that receives oxygen-rich
blood from the left ventricle and sends it through the
body. When the left ventricle and the aorta are too
small, they can't supply the body with enough blood.
Also in HLHS, the mitral and aortic valves are often
narrow or absent. The mitral valve acts like a door that
opens to let blood into the left ventricle. The aortic
valve lets blood out of the left ventricle. Without these
openings, the left ventricle is shut off from the rest of
the heart and the aorta. That means the left ventricle
cannot pump any oxygen-rich blood into the body.
Instead, the oxygen-rich blood returns to the right side
of the heart through an atrial septal defect (ASD), a
hole in the wall that divides the right and left sides of
the heart.
In newborns, the atrial septal defect lets oxygen-rich
blood reach the body because blood can pass from
the right side of the heart and into the aorta, without
going through the left side of the heart. The right side
of the heart pumps blood into the pulmonary artery,
and then a channel called the ductus arteriosus
connects the pulmonary artery to the aorta. The
ductus arteriosus is an important pathway in the fetal
heart, but it closes in the first few days after birth.
Until the ductus closes, oxygen-rich blood enters the
right side of the heart through the atrial septal defect.
The right side of the heart then pumps blood into the
pulmonary artery, and the ductus arteriosus lets blood
flow into the aorta. The aorta carries this oxygen-rich
blood to the body.
This means that for the first few days after birth, the
baby may seem normal because oxygen-rich blood is
reaching the rest of the body. But when that ductus
closes, oxygen-rich blood no longer has a way of
entering the aorta.
When there isn't enough oxygen-rich blood reaching
the body, the baby's skin may turn blue. This is called
cyanosis. Once this happens, the baby needs surgery
or a heart transplant right away.
Taken from Hope for HLHS
Tuesday, February 2, 2010
Heart: Happy Heart Month!
Heart Month means something different to me these days. Since joining the "Heart Mom's" club, I have learned a lot but realize I have a lot yet to learn. I thought I would share a few facts about Congenital Heart Defects (CHD) this month. I'd like to post one every day, but I'm not going to commit to that. :) So we'll see where I end up and we'll learn some new stuff together.
"Most people are unaware that up to 1.3 million American alive today have some form of congenital heart defect and about one-half of these individuals are less than 25 years of age.
At least nine of every 1,000 infants born a year have a heart defect. That’s almost one percent of live-born infants. Sometimes the defect is so mild that there are no outward symptoms. In other cases, it’s so severe that the newborn becomes ill soon after birth. In still other cases, signs and symptoms occur only in later childhood.
In the United States, about 36,000 children are born with a heart defect each year. Most of these children can be helped by surgery even if the defect is severe."
Taken from The American Heart Association
"Most people are unaware that up to 1.3 million American alive today have some form of congenital heart defect and about one-half of these individuals are less than 25 years of age.
At least nine of every 1,000 infants born a year have a heart defect. That’s almost one percent of live-born infants. Sometimes the defect is so mild that there are no outward symptoms. In other cases, it’s so severe that the newborn becomes ill soon after birth. In still other cases, signs and symptoms occur only in later childhood.
In the United States, about 36,000 children are born with a heart defect each year. Most of these children can be helped by surgery even if the defect is severe."
Taken from The American Heart Association
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